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Sorry for the delay - I've been sick.
The definitive test for egg quality is IVF. So repeated IVF cycles with slowly developing embryos is strongly suggestive of an egg "problem". My hunch is that it is a problem with the egg cytoplasm.
Changing medication per se will not change much. All products are basically the same - FSH under different labels. The companies claim that their products are different for one reason or another, but that's all marketing and has little basis in biology. HOWEVER, when different medications are used (usually by different physicians since they all seem to have a favorite), they may be used in a different way resulting in a different rate of follicular growth and/or egg maturation. This may alter the developmental potential of eggs (i.e. cytoplasmic maturation). In your case, I think it is unlikely that changing medications will make a huge difference. I'm sure each of your cycles was a little different (i.e. number of days of stimulation, peak E2, etc) and the development of the embryos was pretty consistent.
Based on existing knowledge, there is no detrimental effect to repeated stimulated cycles. After all, the medications are the same hormones present in your body every month and the medications only act to "salvage" follicles that were recruited in a growing pool but would have died off anyway. You are not depleting you eggs at a faster rate that you would normally. There is no tie between the use of gonadotropins (FSH,LH) and any disease.
It is possible that you and your husband have a genetic problem that resultsin slow embryonic growth, but I think that's a long shot. Although I can't rule out a genetic problem with the sperm, embryonic arrest before Day 4 is almost always caused by cytoplasmic or gentic problems with the egg. Donor sperm and donor egg is always a possibility. It puts you and your husband on equal genetic footing. That is, neither of you is genetically reated to the child. In a sense, its adoption but you are in control of the prenatal care. From a purely biological point of view, I don't think donor sperm is necessary.
PGD tests only the tip of the genetic iceberg. It is a very crude tool. Although embryos can be considered "normal" by PGD, they may still be developmentally compromised.
As a precaution, you could go through immunological testing to determine if there is a problem on the uterine side which may be preventing implantation. Immune testing does not address the slowly developing embryo problem, but if you do decide to go to DE or DE/DS, it would be reassuring to know that implantation will not be an issue.
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